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Volume 6, Issue 6, Supplement, Pages S289-S298 (November 2006)


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Laminoplasty: a review of its role in compressive cervical myelopathy

James J. Hale, MD, Konrad I. Gruson, MD, Jeffrey M. Spivak, MDCorresponding Author Informationemail address

Abstract 

Background context

The currently accepted surgical treatments for compressive cervical myelopathy include both anterior and posterior decompression. Anterior approaches including multilevel discectomy with fusion or vertebral corpectomy with strut grafting, both with and without instrumentation, have enjoyed successful outcomes, but have been associated with select postoperative complications. Laminoplasty has been developed to decompress the spine posteriorly while avoiding the spinal destabilization seen after laminectomy.

Purpose

The purpose of this article is to provide a review of the various techniques, biomechanical basis, predictive value of imaging modalities, clinical outcomes, and postoperative complications associated with cervical laminoplasty.

Study design

A review of the literature.

Methods

A comprehensive literature review using Medline was performed identifying relevant articles that addressed the techniques, clinical outcomes, and complications after cervical laminoplasty, as well as preoperative radiographic predictors of outcome.

Results

The various modifications of cervical laminoplasty have generally been associated with excellent clinical outcomes when used for myelopathy secondary to cervical spondylosis or ossification of the posterior longitudinal ligament (OPLL). Recent long-term studies have identified issues with this technique including axial neck pain, canal restenosis, nerve root palsy, diminished cervical motion, and loss of cervical lordotic alignment.

Conclusions

Cervical laminoplasty remains a reliable procedure for posterior decompression of the spine, but the optimal approach to cervical myelopathy must take into account both patient and disease characteristics, as well as the capabilities and experience of the surgeon.

New York University–Hospital for Joint Diseases, 301 East 17th Street, New York, NY 10003, USA

Corresponding Author InformationCorresponding author. NYU–Hospital for Joint Diseases, 301 East 17th Street, New York, NY 10003. Tel.: (212) 679-1575.

 FDA device/drug status: not applicable.

Nothing of value received from a commercial entity related to this manuscript.

Study conducted at the Hospital for Joint Diseases Orthopaedic Institute, New York, NY.

PII: S1529-9430(06)00199-9

doi:10.1016/j.spinee.2005.12.032


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